| Alzheimer's disease is irreversible. It
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| | highlights the role of the beta-amyloid
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| slowly eats the cognitive functioning of
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| | to the disease. Beta-amyloid is the
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| the people affected by it. The mind
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| | protein fragment- the key component of
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| slowly deteriorates and even the carrying
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| | the AD plaques. Research points out the
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| out of simple tasks may be severely
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| | mutations in any of the three genes
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| affected. The risks of developing this
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| | namely the Chromosome 1, 14 and 21 can
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| brain disease run high by the age of 60
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| | significantly add to the presence of the
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| and older. It is normally characterized
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| | beta-amyloid in the brain. This condition
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| by loss of reason, memory, language
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| | applies for the early onset AD.For the
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| facility and judgment.Contrary to common
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| | late-onset AD however, the Chromosome 19
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| belief, Alzheimer's disease is not an
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| | is known to be playing as a risk factor.
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| element of normal aging. It is a disease
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| | They identified that a certain protein
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| that depreciates the brain. Without this,
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| | named ApoE or apolipoprotein E binds
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| human brain often functions well even up
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| | quickly and tightly to the
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| to an individual's 100th years.Four
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| | beta-amyloid.TauThe tau is a protein
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| million Americans are known to have
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| | normally found in the nerve cells in the
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| Alzheimer's disease and this number is
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| | brain. A person with AD has abnormal
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| expected to rise to 14 million by the
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| | tangled filaments in the
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| year 2050. Given that the current trend
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| | neurons.Cardiovascular Risk FactorsThere
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| of American population is constant and no
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| | are potential links to the causes of
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| advanced treatment is discovered in
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| | cardiovascular diseases and AD according
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| opposition from this disease. The average
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| | to recent studies. One of which is an
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| number of people with Alzheimer's disease
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| | amino acid termed as the homocysteine I
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| doubles every year from 1995 to 2005.
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| | both a risk factor to AD and heart
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| This increase is determined to be the
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| | disease.Oxidative Damage from Free
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| cause of the two playing factors: 1)
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| | RadicalsThe damage in the neurons caused
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| people tend to be prone to risks of
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| | by the build up of too much free radicals
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| getting the Alzheimer's disease as they
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| | is called the oxidative damage. This
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| age and 2) the significant growth of the
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| | process may contribute to the
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| number of older people, especially those
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| | degeneration of the brain by upsetting
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| who age 85 years and older.There are two
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| | the flows of substances from the
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| types of Alzheimer's disease. The FAD or
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| | cells.InflammationThe inflammation factor
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| the Familial Alzheimer's Disease that are
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| | is still debatable. Researchers argue if
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| typically inherited and usually inflicts
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| | inflammation is good or bad for the
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| people aged 30 to 60. The second type is
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| | development of AD. Some thinks that
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| the late-onset Alzheimer's disease is the
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| | inflammation is instrumental to the death
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| more common form that affects people age
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| | of some neurons, some proposes that
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| 65 and more. The cause of Alzheimer's
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| | inflammation is a component in the
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| disease is yet to be revealed. However,
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| | brain's healing process.Brain
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| research points out that it is the result
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| | InfarctionEvidences that people who have
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| of complex events that occur in the brain
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| | experienced stroke in certain brain
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| for certain duration of time. Yet the
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| | regions are likely to develop AD.These
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| present-day concept of how this disease
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| | findings are the current known causes for
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| develops is roughly divided into the
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| | developing the disease. Yet many of
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| following:Genetic Factors and the
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| | which are yet to be researched and
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| Beta-amyloidMutations on the APP or the
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| | studied accordingly.Robert Thatcher is a
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| amyloid precursor protein is squarely
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| | freelance publisher based in Cupertino,
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| defined as one of the causes of the
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| | California.
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| Alzheimer's disease, which then
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|