Mental Health Disorders: Schizophrenia

Schizophrenia affects around 1% of the population ofimplicated in schizophrenia, such as dopamine,
this planet. It is a chronic, severe, and disabling mentalglutamate and GABA. Other genes code for proteins
disorder. It has also been recognized for centuries,involved in the brain development, while others code
maybe not with the same nomenclature, but mostfor proteins of yet undetermined function". (NIMH -
likely with the same symptoms. However, theNational Institute of Mental Health**).
complex mechanisms which cause this conditionFacts and the treatments
remain a mystery of their own…but for howSchizophrenia is a chronic and disabling disorder, but
long?nevertheless misinterpreted by the general population.
History of Schizophrenia: from demons to genesIn fact, between 25 and 40% of people who
The symptoms commonly associated withexperience a psychotic episode not only recover, but
schizophrenia are on the records of many ancientnever experience a second episode. In many
civilizations, such as the Greeks, Romans andinstances, patients enter hospital care voluntarily, and
Egyptians. The causes for this mental disorder haveare able to function normally when not experiencing
been associated with demons, gods, poisonousan episode. Although people with schizophrenia are
substances, dark creatures and more - but untilmore likely to attempt suicide, they are not
today, there are still no definite answers. There isparticularly prone to violence or crimes.
evidence, however, to the different treatmentsOver the last few years, new anti-psychotic
tested (and used) in several patients. Drilling holes in amedications have been developed - such as clozapine,
patient's skull and performing dance rituals wererisperidone and olanzapine. These drugs do not cure
some of them.schizophrenia, but they effectively alleviate its
The term 'Schizophrenia' (derived from the Greeksymptoms and extrapyramidal side effects (such as
words 'schizo'=split and 'phrene'=mind) was onlyrigidity, muscle spasms and tremors) which used to
created in 1911 by Eugene Bleuler, a Swiss physician.appear in older drug treatments.
Bleuler changed the term 'dementia praecox' createdPsychosocial treatment is also important in the
by Emile Kraepelin - the German physician who firstmanagement of schizophrenia. Rehabilitation and
classified mental disorders in categories - because thesubstance abuse treatment are helpful in integrating
disorder was not a dementia. Later on, both scientiststhe patient to the society, and improving the
sub-classified schizophrenia into categories based onpatient's capability to counteract the degenerative
specific symptoms, three of which were delineated ineffects of the disease. Cognitive Behaviour Therapy
the DSM-III (Diagnostic and Statistical Manual of(CBT) can also help. This treatment seems to be
Mental Disorders): disorganized, catatonic, paranoid,effective in reducing the severity of symptoms and
residual, and undifferentiated. Kraepelin's classificationdecreasing the risk of relapse in patients which
remains influential and it is the basis of the DSM-IV.symptoms persist even when they take medication.
Nowadays, it is believed that schizophrenia is causedFurthermore, the efficacy of treatment can be vastly
by a mixture of gene interaction and environmentalimproved with the participation of family and friends.
influence. However, it is not clear how much influenceBecause the positive symptoms of this mental illness
each of these causes wields.are easy to distinguish, both patient and relatives are
The Symptomsable to act when noticing the prevalence of different
The symptoms of schizophrenia are divided intobehaviour.
three categories: positive, negative and cognitive.Overcoming schizophrenia: too far or too close?
Positive symptoms are volatile, easy to recognise andThe future of schizophrenia runs parallel to most
normally involve some loss of contact with reality.research in mental health. With the advent of new
Hallucinations (distortions of the human senses),brain mapping and scanning technologies, such as
delusions (false personal beliefs) and movementPET, MRI, fMRI and the developments in genetic
disorders are examples of such symptoms.research - scientists are narrowing their focus to the
Negative symptoms are harder to recognise as theyinteraction between neurons and how each of our
relate to some loss in abilities such as planning,cognitive functions affect neurotransmission at a
speaking, expressing emotions or being motivated.molecular level. With this in hand, there are
These symptoms can easily be misconnected toexpectations that the approach to schizophrenia will
laziness, depression or other conditions which involvebe more specific and efficient - but to predict cure at
similar behaviour. The term is referred as 'negative'this stage, is a long shot.
due to the reductions in 'normality' - both emotionalNevertheless, there may be more excitement in the
and behavioural.other side of the spectrum. Identifying the exact
Cognitive symptoms relate to problems in normalgene combination which increases the incidence of
cognitive functions. In many cases these cognitivethis condition, and the role of non-genetic factors,
impairments are only detected throughmay still seem distant. However, another solution
neuropsychological tests. Such symptoms includecould effectively help the vast population suffering
problems with memory, attention, decision-makingfrom the symptoms of schizophrenia.
and more.The general thinking is that the best way to combat
The Causesan illness is to develop a cure. What about instead of
As previously noted, schizophrenia is believed to befinding the cure, combating the symptoms? That is
caused by a mixture of gene and environmentalthe approach used for most illnesses which cure is
influence. Davies stated that "schizophrenia appearsyet to be discovered, and it could be a temporary
to be caused by abnormalities in the development ofsolution to improve the lives of millions. Cognitive
the brain that become manifest in late adolescenceenhancers, such as nootropics (cited in a previous
or the early twenties, a time when a differentiationedition of this ezine) and cognitive enhancing methods
and maturation of the central nervous system is at- such as the use of electric waves in specific brain
its most complex" (Davies 2005 p. 210*). However,regions - could counteract some of the debilitating
the disorder is not characterised by a singlesymptoms of schizophrenia.
predominant gene - it relies on the combination of aMost of these techniques are still being tested, but
group of genes, each exerting a small effect. Thesethey are much closer to reality and have already
gene combinations, along with the presencepredictions to reach the market within a few years.
non-genetic factors such as exposure to viruses orAlthough there have been some studies in cognitive
drug abuse, play a key role of increasing vulnerability.enhancing drugs, new drug classes are on the scene -
Current research is attempting to identify the genesand brain science and pharmacology are allowing
which could be directly involved with the incidence offurther discoveries in the field. What will be the next
schizophrenia and psychotic episodes. There arestep? That might not be the one million dollar
candidate genes - however there is not enoughquestion, but perhaps it holds the answer which over
evidence to prove that these genes in fact increase65 million people have been waiting for.
risk. "Some genes that have recently been associatedSources
with schizophrenia code for enzymes and proteins*Davies, J. (November 2005) A Manual of Mental
that help brain cells communicate with each other.Health Care in General Practice, Commonwealth
Some of these enzymes and proteins are involved inDepartment of Health and Ageing, Canberra.
neurotransmitter systems that have long been